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This finding is particularly significant, as CAP is known to bind to the transmembrane domains of TRPV1 channels 60 , suggesting that exendin 9–39 is a selective and potent antagonist for pain relief without competitively displacing CAP at its binding site
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Furthermore, the ileum of germ-free and antibiotic-treated mice showed lower Glp1r gene expression compared to controls, with higher incretin effect resistance (68)
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